Benthiavalicarb inhibits mycelia growth, zoosporangia germination cystospore germination, and sporulation of Phytophthora infestans at very low concentration. CAA mode of action involves inhibition of cell wall synthesis of oomycetes by blocking the activity of cellulose synthase Ces3A. īenthiavalicarb belongs to the carboxylic acid amides (CAA) group of fungicides (FRAC code 40). OXPT+mefenoxam was effective against mefenoxam-resistant isolates of these pathogens. infestans in tomato and Pseudoperonospora cubensis in cucumber. Oxathiapiprolin mixtures (with azoxystrobin, mandipropamid or mefenoxam) were reported to be highly effective against P. Therefore, resistance management is required, including mixing with another fungicide with a different mode of action. Īccording to FRAC ( ), the resistance risk of OXTP is medium to high. Mutants with any of these mutations may have survival potential in the field. These authors reported on three point-mutations that confer resistance to OXPT in P. Resistance against OXPT was induced in Phytophthora capsici by UV irradiation. Oxathiapiprolin is highly effective when applied to the foliage the root system or the seeds. It shows translaminar and acropetally systemic movements. Curatively, it stops mycelial growth, inhibits lesion expansion and inhibits spore production. Preventatively, it inhibits zoospore release, zoospore motility, cystospore germination and direct germination of sporangia. It is extremely active against plant pathogenic oomycetes except Pythium (see literature cited by ). Oxathiapiprolin (OXPT) is a new piperidinyl thiazole isoxazoline fungicide (FRAC code U15) that targets the oxysterol binding proteins in oomycete cells. Thirty-six fungicides and fungicidal mixtures are currently (March 2020) registered in Europe for late blight control ( The recently introduced fungicides are oxathiapiprolin (2017), oxathiapiprolin+famoxadone (2018), oxathiapiprolin+amisulbrom (2018), benthiavalicarb (2018) and oxathiapiprolin+benthiavalicarb (2019). Fungicidal sprays serve as a major measure for combating the disease. Late blight caused by the oomycete Phytophthora infestans (Mont.) De Barry is a devastating disease of potato and tomato world-wide. This shall minimize the risk of developing resistance against either fungicide in the treated crops. The systemic nature of oxathiapiprolin and benthiavalicarb composing ZE assures the translocation to the foliage of two fungicides with different modes of action. The data demonstrate a new strategy for season-long protection of potato against late blight by a single soil application of ZE. Plants treated via the soil were significantly better protected against late blight compared to the plants treated by a spray. Potato plants grown in loess soil in the field were either sprayed or drenched with ZE. Interestingly, oxathiapiprolin persisted in the treated soil for at least 139 days, providing systemic protection against late blight to the following potato crops grown in that treated soils. Artificial inoculations with Phytophthora infestans revealed that such treated plants were protected against late blight in a dose-dependent manner all along the season. In two field experiments, performed in 20, potato plants grown in 64 L containers were treated with a soil drench of oxathiapiprolin, benthiavalicarb or ZE at 12.5, 25 or 50 mg ai/five plants in a container. Here we report that these fungicides can effectively protect potato plants against late blight when applied to the soil in which such potato plants are grown. Root treatment with oxathiapiprolin, benthiavalicarb or their mixture Zorvec-Endavia was shown to provide prolonged systemic protection against foliar oomycete pathogens attacking cucumber, tomato and basil.
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